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The thiol-based antioxidant NAC has undergone clinical trials for use in respiratory related infections (Schito et al., 2007

They proposed that the protein aggregates formed by SQSTM1 during systemic proteasome inhibition sequester mTOR and prevent its TFEB phosphorylation

Nature 500 , 422426 (2013)

Interestingly, in a mouse model of coronary atherosclerosis, hemodynamic stress was shown to induce changes in sphingolipid metabolism that favored the production of S1P over ceramides, suggesting that blood vessels may be adaptative to stress [112]